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with data indicating systemic inflammation or a relapse during treatment&#44;<a class="elsevierStyleCrossRef" href="#bib0075"><span class="elsevierStyleSup">2</span></a> although it is asymptomatic in the majority of patients&#44; or is manifested by means of systemic signs like fever or constitutional symptoms&#44;<a class="elsevierStyleCrossRef" href="#bib0080"><span class="elsevierStyleSup">3</span></a> as observed in our patient&#46;</p><p id="par0010" class="elsevierStylePara elsevierViewall">On the other hand&#44; psoriatic arthritis &#40;PsA&#41; has a prevalence of 0&#46;5&#37;&#44; affecting between 5&#37; and 20&#37; of patients with psoriasis&#46;<a class="elsevierStyleCrossRef" href="#bib0085"><span class="elsevierStyleSup">4</span></a> In a retrospective study&#44; Makredes et al&#46; found that PsA had a higher prevalence ratio associated with GCA&#46;<a class="elsevierStyleCrossRef" href="#bib0090"><span class="elsevierStyleSup">5</span></a> To date&#44; there have been reports of 2 patients with PsA who developed GCA&#46;<a class="elsevierStyleCrossRefs" href="#bib0095"><span class="elsevierStyleSup">6&#44;7</span></a></p></span><span id="sec0010" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0030">Case Report</span><p id="par0015" class="elsevierStylePara elsevierViewall">Our patient was a 65-year-old woman with a history of bronchial asthma&#44; with bronchiectasis&#44; and PsA with mixed erosive involvement&#59; she was negative for HLA-B27&#46; She had received methotrexate and leflunomide from 2011 until 2013&#44; when they were interrupted because of remission&#46; She was presented with a 2-month history of fever and weakness&#44; but did not report headaches&#44; vision disorders or chest&#44; abdominal or limb pain&#46; Physical examination revealed only a difference in arterial blood pressure of 20<span class="elsevierStyleHsp" style=""></span>mmHg and that some pulse rates were lower in left limbs&#46; The temporal arteries were not tender on palpation&#44; and the pulse rate was normal and symmetric&#46; Laboratory tests showed a hemoglobin concentration of 9&#46;9<span class="elsevierStyleHsp" style=""></span>g&#47;dL &#40;normal&#58; 12&#46;0&#8211;15&#46;0&#41;&#44; erythrocyte sedimentation rate of 102<span class="elsevierStyleHsp" style=""></span>mm&#47;h &#40;0&#8211;20&#41;&#44; C-reactive protein 140&#46;90<span class="elsevierStyleHsp" style=""></span>mg&#47;L &#40;&#60;5<span class="elsevierStyleHsp" style=""></span>mg&#47;L&#41; and iron 11<span class="elsevierStyleHsp" style=""></span>&#956;g&#47;dL &#40;35&#46;0&#8211;145&#46;0&#41;&#46; The remaining parameters were normal &#40;thyroid-stimulating hormone&#44; creatine phosphokinase&#44; Mantoux test&#44; tumor markers&#44; protein profile&#44; serological tests&#44; immunological tests&#44; urinary sediment and cultures&#41;&#46; Chest radiography&#44; echocardiogram&#44; gastroscopy with duodenal biopsy and colonoscopy were normal&#46; Chest and abdominal computed tomography &#40;CT&#41; showed stable bronchiectasis and thickening of the abdominal aortic wall&#44; indicative of aortitis&#46; Computed tomography angiography &#40;CTA&#41; &#40;<a class="elsevierStyleCrossRef" href="#fig0005">Fig&#46; 1</a>&#41; also showed evidence of concentric inflammatory thickening of the supra-aortic trunks and aortic arch&#46; Temporal artery biopsy was performed with the histological confirmation of GCA&#46; Treatment was begun with corticoids at a tapering dose starting with 2<span class="elsevierStyleHsp" style=""></span>mg&#47;kg&#47;day and methotrexate at 15<span class="elsevierStyleHsp" style=""></span>mg&#47;weekly&#44; and the symptoms disappeared&#46;</p><elsevierMultimedia ident="fig0005"></elsevierMultimedia></span><span id="sec0015" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0035">Discussion</span><p id="par0020" class="elsevierStylePara elsevierViewall">We present the case of a patient with PsA in remission who developed GCA&#46; Reviewing the literature&#44; we found only 2 other examples&#59; in neither of them was aortic involvement associated with GCA&#46; In 1989&#44; Clementz et al&#46;<a class="elsevierStyleCrossRef" href="#bib0095"><span class="elsevierStyleSup">6</span></a> reported the case of a 63-year-old woman with PsA who developed GCA associated with pericarditis and pancreatic insufficiency&#46; Corli et al&#46;<a class="elsevierStyleCrossRef" href="#bib0100"><span class="elsevierStyleSup">7</span></a> described that of a 62-year-old man with PsA and ulcerative colitis&#44; treated with adalimumab&#44; who developed GCA&#46; They comment on whether anti-tumor necrosis factor &#40;TNF&#41; therapy could act as an inducer&#59; they ultimately conclude that this association cannot be substantiated and appears to be merely a coincidence&#46; Makredes et al&#46;<a class="elsevierStyleCrossRef" href="#bib0090"><span class="elsevierStyleSup">5</span></a> observed that PsA is more frequently associated with the development of other autoimmune diseases&#44; such as inflammatory bowel disease &#40;1&#46;8 &#91;1&#46;3&#8211;2&#46;5&#93;&#41;&#44; pulmonary fibrosis &#40;1&#46;9 &#91;1&#46;2&#8211;3&#46;0&#93;&#41; and&#44; above all&#44; GCA &#40;4&#46;8 &#91;1&#46;5&#8211;15&#46;7&#93;&#41;&#46; This may be due to the fact that certain autoimmune diseases share the same pathogenic pathways&#44; like TNF&#44; which is implicated in the pathogenesis of PsA and GCA&#46; Elkayam et al&#46;<a class="elsevierStyleCrossRef" href="#bib0105"><span class="elsevierStyleSup">8</span></a> reported 5 cases of men diagnosed with ankylosing spondylitis who developed polymyalgia rheumatica &#40;PMR&#41;&#46; Another study showed a higher prevalence of spondyloarthritis &#40;SpA&#41; in patients newly diagnosed with large vessel vasculitis&#44; specifically in 4 of the 15 patients included &#40;6 with GCA and 9 with PMR&#41;&#46; In addition&#44; these patients with concomitant SpA were younger and had higher baseline C-reactive protein levels&#46; The major limitation of that study is the small size of the sample&#44; a circumstance that means that the authors were not able to draw significant conclusions&#46;<a class="elsevierStyleCrossRef" href="#bib0110"><span class="elsevierStyleSup">9</span></a> Recent pathophysiological findings suggest that vasculitis and SpA share a common inflammatory process&#58; the expression of interleukin &#40;IL&#41; 22 was demonstrated in mouse aortic root&#44; as well as in the initial SpA enthesitis&#44; after exposure to IL-23&#46;<a class="elsevierStyleCrossRef" href="#bib0115"><span class="elsevierStyleSup">10</span></a> However&#44; despite the findings of these studies&#44; it is not clear whether there is an association between GCA and chronic SpA&#44; or if they are simply 2 concurrent autoimmune diseases&#46;</p><p id="par0025" class="elsevierStylePara elsevierViewall">On the other hand&#44; aortitis secondary to GCA is a common subclinical entity&#46;<a class="elsevierStyleCrossRef" href="#bib0070"><span class="elsevierStyleSup">1</span></a> In most cases&#44; this inflammatory aortitis is asymptomatic or is only manifested by systemic signs like fever and other constitutional symptoms&#44;<a class="elsevierStyleCrossRef" href="#bib0080"><span class="elsevierStyleSup">3</span></a> as in our patient&#46; For the early diagnosis of aortitis due to GCA&#44; both positron emission tomography &#40;PET&#41; and magnetic resonance imaging are effective&#46;<a class="elsevierStyleCrossRef" href="#bib0075"><span class="elsevierStyleSup">2</span></a> The former using <span class="elsevierStyleSup">18</span>F-fluorodeoxyglucose as a tracer shows an increased metabolic uptake in aorta in approximately 50&#37; of the patients with GCA&#44; enabling direct visualization of the extension of the vascular inflammation&#46; Aortic CT scans provide images of aneurysmal dilatation&#44; ectasia and focal or concentric parietal thickening&#46; Occasionally&#44; abdominal ultrasound makes it possible to visualize the thickening of the vascular wall&#44; with a hypoechoic &#8220;halo&#8221; around the abdominal aorta&#44; which is indicative of aortitis&#46;<a class="elsevierStyleCrossRef" href="#bib0070"><span class="elsevierStyleSup">1</span></a> Some authors propose performing screening for aortic complications based on chest radiography and abdominal ultrasound&#44; possibly completed with CTA at the time of diagnosis&#44; in order to prevent serious complications&#44; like aneurysm rupture or aortic dissection&#46;<a class="elsevierStyleCrossRef" href="#bib0080"><span class="elsevierStyleSup">3</span></a></p><p id="par0030" class="elsevierStylePara elsevierViewall">The differential diagnosis of inflammatory diseases of the aorta comprises a wide spectrum of etiologies from infectious processes to noninfectious disorders like vasculitis&#44; sarcoidosis and IgG4-related lymphoplasmacytic aortitis&#44; and even as a cardiovascular complication secondary to SpA&#46;<a class="elsevierStyleCrossRef" href="#bib0120"><span class="elsevierStyleSup">11</span></a> The literature includes the description of 7 cases of abdominal aortitis due to SpA&#46;<a class="elsevierStyleCrossRefs" href="#bib0125"><span class="elsevierStyleSup">12&#44;13</span></a></p><p id="par0035" class="elsevierStylePara elsevierViewall">In terms of management&#44; the involvement of the aorta and other great vessels in GCA does not change the treatment strategy&#44; which is based initially on corticosteroid therapy&#46;<a class="elsevierStyleCrossRef" href="#bib0075"><span class="elsevierStyleSup">2</span></a></p></span><span id="sec0020" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0040">Conclusions</span><p id="par0040" class="elsevierStylePara elsevierViewall">The case presented here demonstrates the importance of screening for aortic involvement in GCA due to its prognostic and therapeutic implications&#46; Finally&#44; the coexistence between PsA and GCA seems to occur infrequently&#46; At this time&#44; the question as to whether there is an association&#44; or if they simply represent 2 concurrent autoimmune diseases&#44; will need to be evaluated in future studies&#46;</p></span><span id="sec0025" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0045">Ethical Disclosures</span><span id="sec0030" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0050">Protection of human and animal subjects</span><p id="par0045" class="elsevierStylePara elsevierViewall">The authors declare that no experiments were performed on humans or animals for this study&#46;</p></span><span id="sec0035" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0055">Confidentiality of data</span><p id="par0050" class="elsevierStylePara elsevierViewall">The authors declare that they have followed the protocols of their work center on the publication of patient data&#46;</p></span><span id="sec0040" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0060">Right to privacy and informed consent</span><p id="par0055" class="elsevierStylePara elsevierViewall">The authors have obtained the written informed consent of the patients or subjects mentioned in the article&#46; The corresponding author is in possession of this document&#46;</p></span></span><span id="sec0045" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0065">Conflicts of Interest</span><p id="par0060" class="elsevierStylePara elsevierViewall">The authors declare they have no conflicts of interest&#46;</p></span></span>"
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        "resumen" => "<span id="abst0005" class="elsevierStyleSection elsevierViewall"><p id="spar0005" class="elsevierStyleSimplePara elsevierViewall">We report the case of a 65-year-old woman with psoriatic arthritis who developed aortitis secondary to giant cell arteritis&#46; She presented with a 2-month history of dry cough&#44; fever and fatigue&#46; There was no evidence of tumor or infectious processes&#46; Abdominal computed tomographic and computed tomography coronary angiographic findings were suggestive of aortitis&#46; Histological study of a temporal artery biopsy confirmed temporal arteritis&#46; We also review the available literature on this uncommon condition&#46;</p></span>"
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        "resumen" => "<span id="abst0010" class="elsevierStyleSection elsevierViewall"><p id="spar0010" class="elsevierStyleSimplePara elsevierViewall">Presentamos el caso de una mujer de 65 a&#241;os seguida por artritis psori&#225;sica&#44; que desarroll&#243; una aortitis por arteritis de c&#233;lulas gigantes&#44; con un comienzo de tos seca&#44; fiebre y astenia de 2 meses de evoluci&#243;n&#46; Se realiz&#243; estudio descartando procesos tumorales e infecciosos y evidenci&#225;ndose en la TAC abdominal y la angio-TAC datos indicativos de aortitis&#46; Se realiz&#243; biopsia de arteria temporal con confirmaci&#243;n histol&#243;gica de arteritis de c&#233;lulas gigantes&#46; Realizamos una revisi&#243;n de la informaci&#243;n disponible sobre esta infrecuente asociaci&#243;n&#46;</p></span>"
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        "nota" => "<p class="elsevierStyleNotepara" id="npar0005">Please cite this article as&#58; Garc&#237;a-Cez&#243;n de la Cruz MP&#44; Almod&#243;var R&#44; Garc&#237;a P&#233;rez J&#44; Dhimes PF&#44; Zarco P&#46; Aortitis por arteritis de c&#233;lulas gigantes y artritis psori&#225;sica&#58; una asociaci&#243;n infrecuente&#46; Reumatol Clin&#46; 2017&#59;13&#58;230&#8211;232&#46;</p>"
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          "en" => "<p id="spar0015" class="elsevierStyleSimplePara elsevierViewall">Computed tomography angiography&#58; the presence of a concentric inflammatory thickening&#47;vasculitis of all the supra-aortic branches &#40;A&#41; and of the aortic arch &#40;B&#41; is confirmed &#40;arrows&#41;&#46; The greatest involvement appears to be that observed in left subclavian artery&#44; and there is no evidence of significant stenosis at any point&#46; In the carotid territory&#44; only the common carotid artery is affected&#44; there being no extension to internal carotid artery&#46;</p>"
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Case report
Aortitis due to Giant Cell Arteritis and Psoriatic Arthritis: An Uncommon Association
Aortitis por arteritis de células gigantes y artritis psoriásica: una asociación infrecuente
M. del Pilar García-Cezón de la Cruza, Raquel Almodóvarb,
Corresponding author
ralmodovar@fhalcorcon.es

Corresponding author.
, Javier García Pérezc, Patricia Fanny Dhimesd, Pedro Zarcob
a Unidad de Medicina Física y Rehabilitación, Hospital Universitario Fundación Alcorcón, Madrid, Spain
b Unidad de Reumatología, Hospital Universitario Fundación Alcorcón, Madrid, Spain
c Unidad de Radiología Vascular Intervencionista, Hospital Universitario Fundación Alcorcón, Madrid, Spain
d Unidad de Anatomía Patológica, Hospital Universitario Fundación Alcorcón, Madrid, Spain
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    "titulo" => "Aortitis due to Giant Cell Arteritis and Psoriatic Arthritis&#58; An Uncommon Association"
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          "en" => "<p id="spar0015" class="elsevierStyleSimplePara elsevierViewall">Computed tomography angiography&#58; the presence of a concentric inflammatory thickening&#47;vasculitis of all the supra-aortic branches &#40;A&#41; and of the aortic arch &#40;B&#41; is confirmed &#40;arrows&#41;&#46; The greatest involvement appears to be that observed in left subclavian artery&#44; and there is no evidence of significant stenosis at any point&#46; In the carotid territory&#44; only the common carotid artery is affected&#44; there being no extension to internal carotid artery&#46;</p>"
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    "textoCompleto" => "<span class="elsevierStyleSections"><span id="sec0005" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0025">Introduction</span><p id="par0005" class="elsevierStylePara elsevierViewall">Giant cell arteritis &#40;GCA&#41; is a systemic vasculitis involving medium and large vessels&#44; with an annual incidence of 12&#8211;17 cases&#47;100&#44;000 population&#46; It occurs more frequently in women and generally affects individuals over 50 years of age&#46; The branches of the external carotid artery are those most commonly involved&#44; although aortitis can develop in a certain subtype of patient&#46; The involvement of the aorta is probably underestimated&#58; subclinical aortitis has been detected in 20&#37;&#8211;65&#37; of the patients at diagnosis&#46;<a class="elsevierStyleCrossRef" href="#bib0070"><span class="elsevierStyleSup">1</span></a> It should be considered in those with atypical presentations of GCA&#44; with data indicating systemic inflammation or a relapse during treatment&#44;<a class="elsevierStyleCrossRef" href="#bib0075"><span class="elsevierStyleSup">2</span></a> although it is asymptomatic in the majority of patients&#44; or is manifested by means of systemic signs like fever or constitutional symptoms&#44;<a class="elsevierStyleCrossRef" href="#bib0080"><span class="elsevierStyleSup">3</span></a> as observed in our patient&#46;</p><p id="par0010" class="elsevierStylePara elsevierViewall">On the other hand&#44; psoriatic arthritis &#40;PsA&#41; has a prevalence of 0&#46;5&#37;&#44; affecting between 5&#37; and 20&#37; of patients with psoriasis&#46;<a class="elsevierStyleCrossRef" href="#bib0085"><span class="elsevierStyleSup">4</span></a> In a retrospective study&#44; Makredes et al&#46; found that PsA had a higher prevalence ratio associated with GCA&#46;<a class="elsevierStyleCrossRef" href="#bib0090"><span class="elsevierStyleSup">5</span></a> To date&#44; there have been reports of 2 patients with PsA who developed GCA&#46;<a class="elsevierStyleCrossRefs" href="#bib0095"><span class="elsevierStyleSup">6&#44;7</span></a></p></span><span id="sec0010" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0030">Case Report</span><p id="par0015" class="elsevierStylePara elsevierViewall">Our patient was a 65-year-old woman with a history of bronchial asthma&#44; with bronchiectasis&#44; and PsA with mixed erosive involvement&#59; she was negative for HLA-B27&#46; She had received methotrexate and leflunomide from 2011 until 2013&#44; when they were interrupted because of remission&#46; She was presented with a 2-month history of fever and weakness&#44; but did not report headaches&#44; vision disorders or chest&#44; abdominal or limb pain&#46; Physical examination revealed only a difference in arterial blood pressure of 20<span class="elsevierStyleHsp" style=""></span>mmHg and that some pulse rates were lower in left limbs&#46; The temporal arteries were not tender on palpation&#44; and the pulse rate was normal and symmetric&#46; Laboratory tests showed a hemoglobin concentration of 9&#46;9<span class="elsevierStyleHsp" style=""></span>g&#47;dL &#40;normal&#58; 12&#46;0&#8211;15&#46;0&#41;&#44; erythrocyte sedimentation rate of 102<span class="elsevierStyleHsp" style=""></span>mm&#47;h &#40;0&#8211;20&#41;&#44; C-reactive protein 140&#46;90<span class="elsevierStyleHsp" style=""></span>mg&#47;L &#40;&#60;5<span class="elsevierStyleHsp" style=""></span>mg&#47;L&#41; and iron 11<span class="elsevierStyleHsp" style=""></span>&#956;g&#47;dL &#40;35&#46;0&#8211;145&#46;0&#41;&#46; The remaining parameters were normal &#40;thyroid-stimulating hormone&#44; creatine phosphokinase&#44; Mantoux test&#44; tumor markers&#44; protein profile&#44; serological tests&#44; immunological tests&#44; urinary sediment and cultures&#41;&#46; Chest radiography&#44; echocardiogram&#44; gastroscopy with duodenal biopsy and colonoscopy were normal&#46; Chest and abdominal computed tomography &#40;CT&#41; showed stable bronchiectasis and thickening of the abdominal aortic wall&#44; indicative of aortitis&#46; Computed tomography angiography &#40;CTA&#41; &#40;<a class="elsevierStyleCrossRef" href="#fig0005">Fig&#46; 1</a>&#41; also showed evidence of concentric inflammatory thickening of the supra-aortic trunks and aortic arch&#46; Temporal artery biopsy was performed with the histological confirmation of GCA&#46; Treatment was begun with corticoids at a tapering dose starting with 2<span class="elsevierStyleHsp" style=""></span>mg&#47;kg&#47;day and methotrexate at 15<span class="elsevierStyleHsp" style=""></span>mg&#47;weekly&#44; and the symptoms disappeared&#46;</p><elsevierMultimedia ident="fig0005"></elsevierMultimedia></span><span id="sec0015" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0035">Discussion</span><p id="par0020" class="elsevierStylePara elsevierViewall">We present the case of a patient with PsA in remission who developed GCA&#46; Reviewing the literature&#44; we found only 2 other examples&#59; in neither of them was aortic involvement associated with GCA&#46; In 1989&#44; Clementz et al&#46;<a class="elsevierStyleCrossRef" href="#bib0095"><span class="elsevierStyleSup">6</span></a> reported the case of a 63-year-old woman with PsA who developed GCA associated with pericarditis and pancreatic insufficiency&#46; Corli et al&#46;<a class="elsevierStyleCrossRef" href="#bib0100"><span class="elsevierStyleSup">7</span></a> described that of a 62-year-old man with PsA and ulcerative colitis&#44; treated with adalimumab&#44; who developed GCA&#46; They comment on whether anti-tumor necrosis factor &#40;TNF&#41; therapy could act as an inducer&#59; they ultimately conclude that this association cannot be substantiated and appears to be merely a coincidence&#46; Makredes et al&#46;<a class="elsevierStyleCrossRef" href="#bib0090"><span class="elsevierStyleSup">5</span></a> observed that PsA is more frequently associated with the development of other autoimmune diseases&#44; such as inflammatory bowel disease &#40;1&#46;8 &#91;1&#46;3&#8211;2&#46;5&#93;&#41;&#44; pulmonary fibrosis &#40;1&#46;9 &#91;1&#46;2&#8211;3&#46;0&#93;&#41; and&#44; above all&#44; GCA &#40;4&#46;8 &#91;1&#46;5&#8211;15&#46;7&#93;&#41;&#46; This may be due to the fact that certain autoimmune diseases share the same pathogenic pathways&#44; like TNF&#44; which is implicated in the pathogenesis of PsA and GCA&#46; Elkayam et al&#46;<a class="elsevierStyleCrossRef" href="#bib0105"><span class="elsevierStyleSup">8</span></a> reported 5 cases of men diagnosed with ankylosing spondylitis who developed polymyalgia rheumatica &#40;PMR&#41;&#46; Another study showed a higher prevalence of spondyloarthritis &#40;SpA&#41; in patients newly diagnosed with large vessel vasculitis&#44; specifically in 4 of the 15 patients included &#40;6 with GCA and 9 with PMR&#41;&#46; In addition&#44; these patients with concomitant SpA were younger and had higher baseline C-reactive protein levels&#46; The major limitation of that study is the small size of the sample&#44; a circumstance that means that the authors were not able to draw significant conclusions&#46;<a class="elsevierStyleCrossRef" href="#bib0110"><span class="elsevierStyleSup">9</span></a> Recent pathophysiological findings suggest that vasculitis and SpA share a common inflammatory process&#58; the expression of interleukin &#40;IL&#41; 22 was demonstrated in mouse aortic root&#44; as well as in the initial SpA enthesitis&#44; after exposure to IL-23&#46;<a class="elsevierStyleCrossRef" href="#bib0115"><span class="elsevierStyleSup">10</span></a> However&#44; despite the findings of these studies&#44; it is not clear whether there is an association between GCA and chronic SpA&#44; or if they are simply 2 concurrent autoimmune diseases&#46;</p><p id="par0025" class="elsevierStylePara elsevierViewall">On the other hand&#44; aortitis secondary to GCA is a common subclinical entity&#46;<a class="elsevierStyleCrossRef" href="#bib0070"><span class="elsevierStyleSup">1</span></a> In most cases&#44; this inflammatory aortitis is asymptomatic or is only manifested by systemic signs like fever and other constitutional symptoms&#44;<a class="elsevierStyleCrossRef" href="#bib0080"><span class="elsevierStyleSup">3</span></a> as in our patient&#46; For the early diagnosis of aortitis due to GCA&#44; both positron emission tomography &#40;PET&#41; and magnetic resonance imaging are effective&#46;<a class="elsevierStyleCrossRef" href="#bib0075"><span class="elsevierStyleSup">2</span></a> The former using <span class="elsevierStyleSup">18</span>F-fluorodeoxyglucose as a tracer shows an increased metabolic uptake in aorta in approximately 50&#37; of the patients with GCA&#44; enabling direct visualization of the extension of the vascular inflammation&#46; Aortic CT scans provide images of aneurysmal dilatation&#44; ectasia and focal or concentric parietal thickening&#46; Occasionally&#44; abdominal ultrasound makes it possible to visualize the thickening of the vascular wall&#44; with a hypoechoic &#8220;halo&#8221; around the abdominal aorta&#44; which is indicative of aortitis&#46;<a class="elsevierStyleCrossRef" href="#bib0070"><span class="elsevierStyleSup">1</span></a> Some authors propose performing screening for aortic complications based on chest radiography and abdominal ultrasound&#44; possibly completed with CTA at the time of diagnosis&#44; in order to prevent serious complications&#44; like aneurysm rupture or aortic dissection&#46;<a class="elsevierStyleCrossRef" href="#bib0080"><span class="elsevierStyleSup">3</span></a></p><p id="par0030" class="elsevierStylePara elsevierViewall">The differential diagnosis of inflammatory diseases of the aorta comprises a wide spectrum of etiologies from infectious processes to noninfectious disorders like vasculitis&#44; sarcoidosis and IgG4-related lymphoplasmacytic aortitis&#44; and even as a cardiovascular complication secondary to SpA&#46;<a class="elsevierStyleCrossRef" href="#bib0120"><span class="elsevierStyleSup">11</span></a> The literature includes the description of 7 cases of abdominal aortitis due to SpA&#46;<a class="elsevierStyleCrossRefs" href="#bib0125"><span class="elsevierStyleSup">12&#44;13</span></a></p><p id="par0035" class="elsevierStylePara elsevierViewall">In terms of management&#44; the involvement of the aorta and other great vessels in GCA does not change the treatment strategy&#44; which is based initially on corticosteroid therapy&#46;<a class="elsevierStyleCrossRef" href="#bib0075"><span class="elsevierStyleSup">2</span></a></p></span><span id="sec0020" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0040">Conclusions</span><p id="par0040" class="elsevierStylePara elsevierViewall">The case presented here demonstrates the importance of screening for aortic involvement in GCA due to its prognostic and therapeutic implications&#46; Finally&#44; the coexistence between PsA and GCA seems to occur infrequently&#46; At this time&#44; the question as to whether there is an association&#44; or if they simply represent 2 concurrent autoimmune diseases&#44; will need to be evaluated in future studies&#46;</p></span><span id="sec0025" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0045">Ethical Disclosures</span><span id="sec0030" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0050">Protection of human and animal subjects</span><p id="par0045" class="elsevierStylePara elsevierViewall">The authors declare that no experiments were performed on humans or animals for this study&#46;</p></span><span id="sec0035" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0055">Confidentiality of data</span><p id="par0050" class="elsevierStylePara elsevierViewall">The authors declare that they have followed the protocols of their work center on the publication of patient data&#46;</p></span><span id="sec0040" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0060">Right to privacy and informed consent</span><p id="par0055" class="elsevierStylePara elsevierViewall">The authors have obtained the written informed consent of the patients or subjects mentioned in the article&#46; The corresponding author is in possession of this document&#46;</p></span></span><span id="sec0045" class="elsevierStyleSection elsevierViewall"><span class="elsevierStyleSectionTitle" id="sect0065">Conflicts of Interest</span><p id="par0060" class="elsevierStylePara elsevierViewall">The authors declare they have no conflicts of interest&#46;</p></span></span>"
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        "resumen" => "<span id="abst0010" class="elsevierStyleSection elsevierViewall"><p id="spar0010" class="elsevierStyleSimplePara elsevierViewall">Presentamos el caso de una mujer de 65 a&#241;os seguida por artritis psori&#225;sica&#44; que desarroll&#243; una aortitis por arteritis de c&#233;lulas gigantes&#44; con un comienzo de tos seca&#44; fiebre y astenia de 2 meses de evoluci&#243;n&#46; Se realiz&#243; estudio descartando procesos tumorales e infecciosos y evidenci&#225;ndose en la TAC abdominal y la angio-TAC datos indicativos de aortitis&#46; Se realiz&#243; biopsia de arteria temporal con confirmaci&#243;n histol&#243;gica de arteritis de c&#233;lulas gigantes&#46; Realizamos una revisi&#243;n de la informaci&#243;n disponible sobre esta infrecuente asociaci&#243;n&#46;</p></span>"
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Idiomas
Reumatología Clínica (English Edition)
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